Deficiency of complement receptors CR2/CR1 in Cr2⁻/⁻ mice reduces the extent of secondary brain damage after closed head injury.
第一作者:
Miriam D,Neher
第一单位:
Department of Orthopedic Surgery, Denver Health Medical Center, University of Colorado School of Medicine, 777 Bannock Street, Denver CO 80204, USA. philip.stahel@dhha.org.
作者:
医学主题词
动物(Animals);星形细胞(Astrocytes);脑(Brain);补体C3(Complement C3);颅脑损伤(Craniocerebral Trauma);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);同源盒结构域蛋白质类(Homeodomain Proteins);免疫球蛋白M(Immunoglobulin M);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);小神经胶质细胞(Microglia);神经元(Neurons);磷酸丙酮酸水合酶(Phosphopyruvate Hydratase);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);受体, 补体3b(Receptors, Complement 3b);受体, 补体3d(Receptors, Complement 3d)
DOI
10.1186/1742-2094-11-95
PMID
24885042
发布时间
2021-10-21
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