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首页> Blood> Aberrant overexpression of CD14 on granulocytes sensitizes the innate immune response in mDia1 heterozygous del(5q) MDS.

Aberrant overexpression of CD14 on granulocytes sensitizes the innate immune response in mDia1 heterozygous del(5q) MDS.

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作者： Ganesan,Keerthivasan ^[1] ; Yang,Mei ^[1] ; Baobing,Zhao ^[1] ; Ling,Zhang ^[2] ; Chad E,Harris ^[3] ; Juehua,Gao ^[1] ; Ashley A,Basiorka ^[2] ; Matthew J,Schipma ^[4] ; James,McElherne ^[1] ; Jing,Yang ^[1] ; Amit K,Verma ^[5] ; Andrea,Pellagatti ^[6] ; Jacqueline,Boultwood ^[6] ; Alan F,List ^[2] ; David A,Williams ^[3] ; Peng,Ji ^[1]

作者单位： Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, IL; ^[1] Department of Hematopathology and Laboratory Medicine and Department of Malignant Hematology, H. Lee Moffitt Cancer Center and Research Institute, Tampa, FL; ^[2] Boston Children's Hospital and Dana-Farber Cancer Institute, Harvard Medical School, Harvard Stem Cell Institute, Boston, MA; ^[3] Center for Genetic Medicine, Northwestern University, Chicago, IL; ^[4] Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, NY; and. ^[5] Nuffield Division of Clinical Laboratory Sciences, John Radcliffe Hospital, Oxford, United Kingdom. ^[6]

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);骨髓细胞(Bone Marrow Cells);载体蛋白质类(Carrier Proteins);染色体缺失(Chromosome Deletion);染色体, 人, 5对(Chromosomes, Human, Pair 5);女(雌)性(Female);基因表达调控(Gene Expression Regulation);粒细胞(Granulocytes);杂合子(Heterozygote);人类(Humans);免疫, 先天(Immunity, Innate);脂多糖类(Lipopolysaccharides);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);骨髓增生异常综合征(Myelodysplastic Syndromes);RNA, 信使(RNA, Messenger);Toll样受体4(Toll-Like Receptor 4)

DOI 10.1182/blood-2014-01-552463

PMID 24891322

发布时间 2021-10-21

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Blood

Blood

2014年124卷5期

780-90页

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