Vascular risk and Aβ interact to reduce cortical thickness in AD vulnerable brain regions.

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作者： Sylvia,Villeneuve ^[1] ; Bruce R,Reed ^[2] ; Cindee M,Madison ^[2] ; Miranka,Wirth ^[2] ; Natalie L,Marchant ^[2] ; Stephen,Kriger ^[2] ; Wendy J,Mack ^[2] ; Nerses,Sanossian ^[2] ; Charles,DeCarli ^[2] ; Helena C,Chui ^[2] ; Michael W,Weiner ^[2] ; William J,Jagust ^[2]

作者单位： From the Helen Wills Neuroscience Institute (S.V., C.M.M., M.W., W.J.J.), University of California Berkeley; Department of Neurology (B.R.R., C.D.), University of California Davis; Department of Old Age Psychiatry (N.L.M.), Institute of Psychiatry, King's College London, UK; Center for Imaging of Neurodegenerative Diseases (S.K., M.W.W.), University of California San Francisco; and Departments of Preventive Medicine (W.J.M.) and Neurology (N.S., H.C.C.), University of Southern California, Los Angeles. sylvia.villeneuve@berkeley.edu. ^[1] From the Helen Wills Neuroscience Institute (S.V., C.M.M., M.W., W.J.J.), University of California Berkeley; Department of Neurology (B.R.R., C.D.), University of California Davis; Department of Old Age Psychiatry (N.L.M.), Institute of Psychiatry, King's College London, UK; Center for Imaging of Neurodegenerative Diseases (S.K., M.W.W.), University of California San Francisco; and Departments of Preventive Medicine (W.J.M.) and Neurology (N.S., H.C.C.), University of Southern California, Los Angeles. ^[2]