Gold nanoparticles inhibit VEGF165-induced migration and tube formation of endothelial cells via the Akt pathway.

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作者： Yunlong,Pan ^[1] ; Qing,Wu ^[1] ; Li,Qin ^[2] ; Jiye,Cai ^[3] ; Bin,Du ^[4]

作者单位： Department of General Surgery, The First Affiliated Hospital of Jinan University, Guangzhou 510632, China. ^[1] Department of Histology and Embryology, Medical School of Jinan University, Guangzhou 510632, China. ^[2] Department of Chemistry, Life Science and Technology School of Jinan University, Guangzhou 510632, China. ^[3] Department of Pathology, Medical School of Jinan University, Guangzhou 510632, China ; Division of Clinic Pathology, The First Affiliated Hospital of Jinan University, Guangzhou 510632, China. ^[4]

主题词动物(Animals);毛细血管(Capillaries);细胞运动(Cell Movement);鸡胚(Chick Embryo);金(Gold);人类(Humans);金属纳米粒子(Metal Nanoparticles);新生血管化, 生理性(Neovascularization, Physiologic);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction);血管内皮生长因子A(Vascular Endothelial Growth Factor A);伤口愈合(Wound Healing)

DOI 10.1155/2014/418624

PMID 24987682

发布时间 2022-03-21

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BioMed research international

2014年2014卷

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Gold nanoparticles inhibit VEGF165-induced migration and tube formation of endothelial cells via the Akt pathway.

BioMed research international

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Gold nanoparticles inhibit VEGF165-induced migration and tube formation of endothelial cells via the Akt pathway.

BioMed research international

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BioMed research international

相关期刊

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