Granulovacuolar degeneration and unfolded protein response in mouse models of tauopathy and Aβ amyloidosis.-论文-万方医学网

作者： Christoph,Köhler ^[1] ; Maja,Dinekov ^[2] ; Jürgen,Götz ^[3]

作者单位： Institute II for Anatomy, Medical Faculty, University of Cologne, Kerpener Str. 62, 50924 Cologne, Germany. Electronic address: c.koehler@uni-koeln.de. ^[1] Institute II for Anatomy, Medical Faculty, University of Cologne, Kerpener Str. 62, 50924 Cologne, Germany. ^[2] Clem Jones Centre for Ageing Dementia Research (CADR), Queensland Brain Institute (QBI), The University of Queensland, Brisbane (St Lucia Campus), QLD 4072, Australia. ^[3]

主题词年龄因素(Age Factors);淀粉样β肽类(Amyloid beta-Peptides);淀粉样β蛋白前体(Amyloid beta-Protein Precursor);方差分析(Analysis of Variance);动物(Animals);酪蛋白激酶Ⅰβ(Casein Kinase Idelta);脑淀粉样血管病(Cerebral Amyloid Angiopathy);疾病模型, 动物(Disease Models, Animal);人类(Humans);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);突变(Mutation);神经变性(Nerve Degeneration);神经原纤维缠结(Neurofibrillary Tangles);磷酰化(Phosphorylation);早老素1(Presenilin-1);Tau病变(Tauopathies);非折叠蛋白质应答(Unfolded Protein Response);空泡(Vacuoles);eIF-2激酶(eIF-2 Kinase);tau蛋白质类(tau Proteins)

DOI 10.1016/j.nbd.2014.07.006

PMID 25073087

发布时间 2016-11-25

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Granulovacuolar degeneration and unfolded protein response in mouse models of tauopathy and Aβ amyloidosis.

Neurobiology of disease

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Neurobiology of disease

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Granulovacuolar degeneration and unfolded protein response in mouse models of tauopathy and Aβ amyloidosis.

Neurobiology of disease

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Neurobiology of disease

相关期刊

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