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首页> Physiological genomics> Bioinformatic approaches to augment study of epithelial-to-mesenchymal transition in lung cancer.

Bioinformatic approaches to augment study of epithelial-to-mesenchymal transition in lung cancer.

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作者： Tim N,Beck ^[1] ; Adaeze J,Chikwem ^[2] ; Nehal R,Solanki ^[3] ; Erica A,Golemis ^[4]

作者单位： Developmental Therapeutics Program, Fox Chase Cancer Center, Philadelphia, Pennsylvania; Program in Molecular and Cell Biology and Genetics, Drexel University College of Medicine, Philadelphia, Pennsylvania; and. ^[1] Developmental Therapeutics Program, Fox Chase Cancer Center, Philadelphia, Pennsylvania; Temple University School of Medicine, Philadelphia, Pennsylvania; and. ^[2] Immune Cell Development and Host Defense Program, Fox Chase Cancer Center, Philadelphia, Pennsylvania; Program in Microbiology and Immunology, Drexel University College of Medicine, Philadelphia, Pennsylvania. ^[3] Developmental Therapeutics Program, Fox Chase Cancer Center, Philadelphia, Pennsylvania; Temple University School of Medicine, Philadelphia, Pennsylvania; and Program in Molecular and Cell Biology and Genetics, Drexel University College of Medicine, Philadelphia, Pennsylvania; and Erica.Golemis@fccc.edu. ^[4]

关键词 E-cadherin EMT NEDD9/HEF1 SRC TGF-β bioinformatics cancer epithelial-to-mesenchymal transition genomics precision oncology proteomics sequencing β-catenin

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);钙黏着糖蛋白类(Cadherins);计算生物学(Computational Biology);上皮-间质转化(Epithelial-Mesenchymal Transition);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);肺肿瘤(Lung Neoplasms);模型, 生物学(Models, Biological);磷蛋白类(Phosphoproteins);信号传导(Signal Transduction);转化生长因子β1(Transforming Growth Factor beta1);β连环素(beta Catenin)

DOI 10.1152/physiolgenomics.00062.2014

PMID 25096367

发布时间 2023-11-04

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Physiological genomics

Physiological genomics

2014年46卷19期

699-724页

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