ACAT1-associated Late Endosomes/Lysosomes Significantly Improve Impaired Intracellular Cholesterol Metabolism and the Survival of Niemann-Pick Type C Mice.

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作者： Masashi,Kamikawa ^[1] ; XiaoFeng,Lei ^[2] ; Yukio,Fujiwara ^[3] ; Kazuchika,Nishitsuji ^[4] ; Hiroshi,Mizuta ^[5] ; Motohiro,Takeya ^[3] ; Naomi,Sakashita ^[6]

作者单位： Department of Cell Pathology, Graduate School of Medical Sciences, Kumamoto University ; Department of Orthopaedic Surgery, Graduate School of Medical Sciences, Kumamoto University, 1-1-1 Honjo, Kumamoto 860-8556, Kumamoto, Japan. ^[1] Department of Biochemistry, Showa University School of Medicine, 1-5-8 Hatanodai, Shinagawa-ku, Tokyo 142-8555, Tokyo, Japan. ^[2] Department of Cell Pathology, Graduate School of Medical Sciences, Kumamoto University. ^[3] Department of Human Pathology, Institute of Health Biosciences, The University of Tokushima Graduate School, 3-18-15 Kuramoto-cho, Tokushima 770-8503, Tokushima, Japan. ^[4] Department of Orthopaedic Surgery, Graduate School of Medical Sciences, Kumamoto University, 1-1-1 Honjo, Kumamoto 860-8556, Kumamoto, Japan. ^[5] Department of Cell Pathology, Graduate School of Medical Sciences, Kumamoto University ; Department of Human Pathology, Institute of Health Biosciences, The University of Tokushima Graduate School, 3-18-15 Kuramoto-cho, Tokushima 770-8503, Tokushima, Japan. ^[6]

关键词 Niemann-Pick type C disease acyl-coenzyme A: cholesterol acyltransferase 1 cholesterol late endosomes methyl-β-cyclodextrin

DOI 10.1267/ahc.13033

PMID 25221362

发布时间 2021-10-21

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