GSK3β-dependent inhibition of AMPK potentiates activation of neutrophils and macrophages and enhances severity of acute lung injury.
第一作者:
Dae Won,Park
第一单位:
Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama; Division of Infectious Diseases, Korea University Ansan Hospital, Ansan, Korea;
作者:
医学主题词
AMP活化蛋白激酶类(AMP-Activated Protein Kinases);急性肺损伤(Acute Lung Injury);动物(Animals);细胞系(Cell Line);趋化因子CXCL2(Chemokine CXCL2);色酮类(Chromones);酶抑制剂(Enzyme Inhibitors);糖原合成酶激酶3(Glycogen Synthase Kinase 3);HMGB1蛋白质(HMGB1 Protein);I-κB激酶(I-kappa B Kinase);吲哚类(Indoles);脂多糖类(Lipopolysaccharides);巨噬细胞活化(Macrophage Activation);巨噬细胞(Macrophages);马来酰亚胺类(Maleimides);小鼠(Mice);吗啉类(Morpholines);嗜中性白细胞激活(Neutrophil Activation);中性白细胞(Neutrophils);磷酰化(Phosphorylation);肺水肿(Pulmonary Edema);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1152/ajplung.00165.2014
PMID
25239914
发布时间
2022-12-14
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