Receptor-interacting protein kinase 4 and interferon regulatory factor 6 function as a signaling axis to regulate keratinocyte differentiation.

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作者： Mei Qi,Kwa ^[1] ; Jennifer,Huynh ^[1] ; Jiamin,Aw ^[2] ; Lianyi,Zhang ^[2] ; Thao,Nguyen ^[3] ; Eric C,Reynolds ^[2] ; Matthew J,Sweet ^[4] ; John A,Hamilton ^[3] ; Glen M,Scholz ^[5]

作者单位： From the Oral Health Cooperative Research Centre, Melbourne Dental School, and Bio21 Institute, and Department of Medicine, Royal Melbourne Hospital, The University of Melbourne, Melbourne, Victoria 3010, Australia and. ^[1] From the Oral Health Cooperative Research Centre, Melbourne Dental School, and Bio21 Institute, and. ^[2] Department of Medicine, Royal Melbourne Hospital, The University of Melbourne, Melbourne, Victoria 3010, Australia and. ^[3] the Institute for Molecular Bioscience and Australian Infectious Disease Research Centre, The University of Queensland, Brisbane, Queensland 4072, Australia. ^[4] From the Oral Health Cooperative Research Centre, Melbourne Dental School, and Bio21 Institute, and Department of Medicine, Royal Melbourne Hospital, The University of Melbourne, Melbourne, Victoria 3010, Australia and glenms@unimelb.edu.au. ^[5]

关键词 Differentiation Grainyhead-like 3 Interferon Regulatory Factor 6 Keratinocyte OVO-like 1 Popliteal Pterygium Protein Kinase C (PKC)Protein Phosphorylation Receptor-interacting Protein Kinase 4 Transcription Factor