Deletion of interleukin-6 prevents cardiac inflammation, fibrosis and dysfunction without affecting blood pressure in angiotensin II-high salt-induced hypertension.-论文-万方医学网

作者： Germán E,González ^[1] ; Nour-Eddine,Rhaleb ; Martin A,D'Ambrosio ; Pablo,Nakagawa ; Yunhe,Liu ; Pablo,Leung ; Xiangguo,Dai ; Xiao-Ping,Yang ; Edward L,Peterson ; Oscar A,Carretero

作者单位： aDepartment of Internal Medicine, Hypertension and Vascular Research Division, Henry Ford Hospital, Detroit, Michigan bCardiovascular Pathophysiology Institute, University of Buenos Aires, Argentina cDepartment of Physiology dDepartment of Internal Medicine, Wayne State University eDepartment of Public Health Sciences, Henry Ford Hospital, Detroit, Michigan, USA. ^[1]

主题词白蛋白类(Albumins);血管紧张素Ⅱ(Angiotensin II);动物(Animals);血压(Blood Pressure);心脏扩大(Cardiomegaly);超声心动描记术(Echocardiography);纤维化(Fibrosis);心脏(Heart);心率(Heart Rate);高血压(Hypertension);炎症(Inflammation);白细胞介素6(Interleukin-6);巨噬细胞(Macrophages);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);心肌(Myocardium);肌细胞, 心脏(Myocytes, Cardiac);表型(Phenotype);氯化钠, 膳食(Sodium Chloride, Dietary)

DOI 10.1097/HJH.0000000000000358

PMID 25304471

发布时间 2022-03-11

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Deletion of interleukin-6 prevents cardiac inflammation, fibrosis and dysfunction without affecting blood pressure in angiotensin II-high salt-induced hypertension.

Journal of hypertension

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Journal of hypertension

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Deletion of interleukin-6 prevents cardiac inflammation, fibrosis and dysfunction without affecting blood pressure in angiotensin II-high salt-induced hypertension.

Journal of hypertension

相似文献

Journal of hypertension

相关期刊

检索历史清除