uPAR induces expression of transforming growth factor β and interleukin-4 in cancer cells to promote tumor-permissive conditioning of macrophages.
第一作者:
Jingjing,Hu
第一单位:
Department of Pathology, University of California San Diego School of Medicine, La Jolla, California.
作者:
主题词
动物(Animals);精氨酸酶(Arginase);脑肿瘤(Brain Neoplasms);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);共同培养技术(Coculture Techniques);疾病恶化(Disease Progression);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);胶质母细胞瘤(Glioblastoma);人类(Humans);炎症(Inflammation);白细胞介素4(Interleukin-4);巨噬细胞(Macrophages);小鼠(Mice);肿瘤转移(Neoplasm Metastasis);胰腺肿瘤(Pancreatic Neoplasms);表型(Phenotype);受体, 尿激酶型纤溶酶原激活物(Receptors, Urokinase Plasminogen Activator);信号传导(Signal Transduction);转化生长因子β(Transforming Growth Factor beta)
DOI
10.1016/j.ajpath.2014.08.003
PMID
25310970
发布时间
2022-03-10
- 浏览14
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文