Necrostatin-1 protects against reactive oxygen species (ROS)-induced hepatotoxicity in acetaminophen-induced acute liver failure.
第一作者:
Kenji,Takemoto
第一单位:
Innovation Center for Immunoregulation and Therapeutics, Graduate School of Medicine, Kyoto University, Yoshida Konoe, Sakyo-ku, Kyoto 606-8501, Japan ; Division of Hepato-Biliary-Pancreatic Surgery and Transplantation, Department of Surgery, Graduate School of Medicine, Kyoto University, 54 Kawaharacho, Syogoin, Sakyo-ku, Kyoto 606-8507, Japan.
作者:
关键词
ABTS, 2,2′-azino-bis (3-ethylbenzothiazoline)-6-sulfonic acidALF, acute liver failureALT, alanine aminotransferaseAPAP, acetaminophenAST, aspartate aminotransferaseAcetaminophenAcute liver failureCM-H2DCFDA, 5-(and-6)-chloromethyl-2′,7′-dichlorodihydrofluorescein diacetate, acetyl esterCXCL1, chemokine (C-X-C motif) ligand 1CYP2E1, cytochrome P450 2E1DMSO, dimethyl sulfoxideDrp1, dynamin-related protein 1FBS, fetal bovine serumGSH, glutathioneHepatocytesLDH, lactate dehydrogenaseNAPQI, N-acetyl-p-benzoquinoneNO, nitric oxideNec-1, necrostatin-1NecroptosisPGAM5, phosphoglycerate mutase family member 5PI, propidium iodideRIPK, receptor-interacting protein kinaseRIPK-dependent necrosisROS, reactive oxygen speciesReactive oxygen speciesSNAP, S-nitroso-N-acetyl-dl-penicillamineWST-8, 2-(2-methoxy-4-nitrophenyl)-3-(4-nitrophenyl)-5-(2,4-disulfophenyl)-2H-tetrazoliumbFGF, basic fibroblast growth factorλPP, lambda protein phosphatase
DOI
10.1016/j.fob.2014.08.007
PMID
25349782
发布时间
2021-10-21
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FEBS open bio
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