Alteration of Scn3a expression is mediated via CpG methylation and MBD2 in mouse hippocampus during postnatal development and seizure condition.

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作者： Hai-Jun,Li ^[1] ; Rui-Ping,Wan ^[2] ; Ling-Jia,Tang ^[2] ; Shu-Jing,Liu ^[2] ; Qi-Hua,Zhao ^[2] ; Mei-Mei,Gao ^[2] ; Yong-Hong,Yi ^[2] ; Wei-Ping,Liao ^[2] ; Xiao-Fang,Sun ^[3] ; Yue-Sheng,Long ^[4]

作者单位： Key Laboratory of Neurogenetics and Channelopathies of Guangdong Province and the Ministry of Education of China, Collaborative Innovation Center for Neurogenetics and Channelopathies, Institute of Neuroscience, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, China; Key laboratory of Reproduction and Genetics of Guangdong Higher Education Institutes, Guangzhou Medical University, Guangzhou, China. ^[1] Key Laboratory of Neurogenetics and Channelopathies of Guangdong Province and the Ministry of Education of China, Collaborative Innovation Center for Neurogenetics and Channelopathies, Institute of Neuroscience, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, China. ^[2] Key laboratory of Reproduction and Genetics of Guangdong Higher Education Institutes, Guangzhou Medical University, Guangzhou, China. Electronic address: xiaofangsun@hotmail.com. ^[3] Key Laboratory of Neurogenetics and Channelopathies of Guangdong Province and the Ministry of Education of China, Collaborative Innovation Center for Neurogenetics and Channelopathies, Institute of Neuroscience, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, China. Electronic address: longyuesheng@gzhmu.edu.cn. ^[4]

关键词 CpG methylation MBD2 Promoter Scn3a Seizure