换一批
换一批Autophagy is not required to sustain exercise and PRKAA1/AMPK activity but is important to prevent mitochondrial damage during physical activity.
第一作者：
Francesca,Lo Verso
第一单位：
a Dulbecco Telethon Institute; Venetian Institute of Molecular Medicine ; Padova , Italy.
作者：
关键词
ACACA, acetyl-CoA carboxylase alphaAMPKATG7, autophagy-related 7BNIP3, BCL2/adenovirus E1B 19?kDa interacting protein 3FDB, flexor digitorum brevisMAP1LC3A, microtubule-associated protein 1 light chain 3NAC, N-acetylcysteinePARK2, parkin RBR E3 ubiquitin protein ligasePRKAA1, protein kinase AMP-activated, alpha 1 catalytic subunitROS, reactive oxygen speciesSQSTM1, sequestosome 1TA, tibialis anteriorTMRM, tetramethylrhodamine, methyl esterautophagyexercisemetabolismmitochondriaskeletal muscle
主题词
AMP活化蛋白激酶类(AMP-Activated Protein Kinases);动物(Animals);抗氧化剂(Antioxidants);自噬(Autophagy);女(雌)性(Female);葡萄糖(Glucose);内环境稳定(Homeostasis);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);微管相关蛋白质类(Microtubule-Associated Proteins);线粒体(Mitochondria);肌收缩(Muscle Contraction);氧化性应激(Oxidative Stress);身体条件作用, 动物(Physical Conditioning, Animal)
DOI
10.4161/auto.32154
PMID
25483961
发布时间
2025-01-03
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Autophagy
1883-94页
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