换一批The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid.
第一作者：
Birgit,Steinmetz
第一单位：
a Department of Medicine I ; Medical University of Vienna ; Währinger Gürtel, Vienna , Austria.
作者：
关键词
AML, acute myeloid leukemiaAPL, acute promyelocytic leukemiaATRA, all-trans retinoic acidAr, ATRA regulationDMSO, dimethyl sulfoxideEVI1Em, EVI1 modulationEr, EVI1 regulationFBS, fetal bovine serumFC, fold changeFDR, false discovery rateGDF15GFP, green fluorescent proteinMDS, myelodysplastic syndromePSG, penicillin streptomycin glutamineRAR, retinoic acid receptorRARE, retinoic acid response elementSE, standard errorall-trans retinoic acidapoptosiscell cyclegene expression profilingmcoEvi1, murine codon optimized Evi1myeloid differentiation
主题词
细胞凋亡(Apoptosis);细胞分化(Cell Differentiation);DNA结合蛋白质类(DNA-Binding Proteins);减量调节(Down-Regulation);上皮细胞(Epithelial Cells);基因敲低技术(Gene Knockdown Techniques);生长分化因子15(Growth Differentiation Factor 15);HL-60细胞(HL-60 Cells);人类(Humans);髓系细胞(Myeloid Cells);癌基因(Oncogenes);原癌基因(Proto-Oncogenes);结果可重复性(Reproducibility of Results);转录因子(Transcription Factors);转录, 遗传(Transcription, Genetic);维甲酸(Tretinoin)
DOI
10.4161/15384101.2014.946869
PMID
25486480
发布时间
2020-09-30
- 浏览95
Cell cycle (Georgetown, Tex.)
2931-43页
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