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首页> Nature cell biology> Degradation of Cep68 and PCNT cleavage mediate Cep215 removal from the PCM to allow centriole separation, disengagement and licensing.

Degradation of Cep68 and PCNT cleavage mediate Cep215 removal from the PCM to allow centriole separation, disengagement and licensing.

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作者： Julia K,Pagan ^[1] ; Antonio,Marzio ^[1] ; Mathew J K,Jones ^[2] ; Anita,Saraf ^[3] ; Prasad V,Jallepalli ^[2] ; Laurence,Florens ^[3] ; Michael P,Washburn ^[4] ; Michele,Pagano ^[5]

作者单位： Department of Pathology, Laura and Isaac Perlmutter Cancer Center, New York University School of Medicine, New York 10065, USA. ^[1] Molecular Biology Program, Memorial Sloan-Kettering Cancer Center, 1275 York Avenue New York 10065, USA. ^[2] The Stowers Institute of Medical Research, 1000 East 50th Street, Kansas City Missouri 64110, USA. ^[3] 1] The Stowers Institute of Medical Research, 1000 East 50th Street, Kansas City Missouri 64110, USA [2] Department of Pathology and Laboratory Medicine, The University of Kansas Medical Center, 3901 Rainbow Boulevard, Kansas City Kansas 66160, USA. ^[4] 1] Department of Pathology, Laura and Isaac Perlmutter Cancer Center, New York University School of Medicine, New York 10065, USA [2] Howard Hughes Medical Institute, 522 First Avenue New York 10016, USA. ^[5]

主题词抗原(Antigens);细胞周期蛋白质类(Cell Cycle Proteins);细胞系, 肿瘤(Cell Line, Tumor);中心粒(Centrioles);HEK293细胞(HEK293 Cells);人类(Humans);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);中期(Metaphase);微管相关蛋白质类(Microtubule-Associated Proteins);神经组织蛋白质类(Nerve Tissue Proteins);磷酰化(Phosphorylation);前中期(Prometaphase);蛋白质结合(Protein Binding);原癌基因蛋白质类(Proto-Oncogene Proteins);RNA干扰(RNA Interference);RNA, 小分子干扰(RNA, Small Interfering);SKP Cullin F-框蛋白质连接酶类(SKP Cullin F-Box Protein Ligases)

DOI 10.1038/ncb3076

PMID 25503564

发布时间 2024-09-22

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Nature cell biology

Nature cell biology

2015年17卷1期

31-43页

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