Brain ACE2 overexpression reduces DOCA-salt hypertension independently of endoplasmic reticulum stress.
第一作者:
Huijing,Xia
第一单位:
Department of Pharmacology and Experimental Therapeutics, Cardiovascular Center of Excellence and Neurosciences Center of Excellence, Louisiana State University Health Sciences Center, New Orleans, Louisiana hxia1@lsuhsc.edu.
作者:
医学主题词
动物(Animals);血压(Blood Pressure);脑(Brain);疾病模型, 动物(Disease Models, Animal);内质网(Endoplasmic Reticulum);基因敲低技术(Gene Knockdown Techniques);人类(Humans);高血压(Hypertension);输注, 脑室内(Infusions, Intraventricular);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);下丘脑室旁核(Paraventricular Hypothalamic Nucleus);肽基二肽酶A(Peptidyl-Dipeptidase A);氯化钠, 膳食(Sodium Chloride, Dietary);穹隆下器官(Subfornical Organ);牛磺鹅脱氧胆酸(Taurochenodeoxycholic Acid);时间因素(Time Factors);增量调节(Up-Regulation)
DOI
10.1152/ajpregu.00366.2014
PMID
25519733
发布时间
2024-05-12
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