μ-Opioid and N-methyl-D-aspartate receptors in the amygdala contribute to minocycline-induced potentiation of morphine analgesia in rats.
第一作者:
Hamed,Ghazvini
第一单位:
aDepartment of Animal Biology, School of Biology and Center of Excellence in Phylogeny of Living Organisms, College of Science, University of Tehran bDepartment of Neuroscience, School of Advanced Technologies in Medicine cDepartment of Pharmacology, School of Medicine, Tehran University of Medical Sciences dSchool of Cognitive Sciences, Institute for Research in Fundamental Sciences (IPM), Tehran, Iran.
作者:
主题词
杏仁核(Amygdala);镇痛药(Analgesics);动物(Animals);疾病模型, 动物(Disease Models, Animal);剂量效应关系, 药物(Dose-Response Relationship, Drug);药物疗法, 联合(Drug Therapy, Combination);兴奋性氨基酸激动剂(Excitatory Amino Acid Agonists);热温度(Hot Temperature);注射, 腹腔内(Injections, Intraperitoneal);男(雄)性(Male);米诺环素(Minocycline);吗啡(Morphine);N-甲基天冬氨酸(N-Methylaspartate);纳洛酮(Naloxone);麻醉品拮抗药(Narcotic Antagonists);疼痛(Pain);疼痛测定(Pain Measurement);随机分配(Random Allocation);大鼠, Wistar(Rats, Wistar);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);受体, 阿片样, μ(Receptors, Opioid, mu)
DOI
10.1097/FBP.0000000000000126
PMID
25563202
发布时间
2015-04-27
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Behavioural pharmacology
383-92页
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