In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model.

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作者： Lucia,Hipólito ^[1] ; Amanda K,Fakira ; David,Cabañero ; Rebecca,Blandón ; Susan M,Carlton ; Jose A,Morón ; Zara,Melyan

作者单位： Department of Anesthesiology, Columbia University Medical Center, New York, NY, USA Department of Neuroscience and Cell Biology, University of Texas Medical Branch, Galveston, TX, USA. ^[1]

主题词动物(Animals);疾病模型, 动物(Disease Models, Animal);Freund佐剂(Freund's Adjuvant);吲哚类(Indoles);炎症(Inflammation);注射, 脊髓(Injections, Spinal);男(雄)性(Male);肟类(Oximes);疼痛(Pain);痛阈(Pain Threshold);后角细胞(Posterior Horn Cells);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);小电导钙激活钾通道(Small-Conductance Calcium-Activated Potassium Channels);脊髓(Spinal Cord);治疗结果(Treatment Outcome)

DOI 10.1097/j.pain.0000000000000124

PMID 25734988

发布时间 2022-10-05

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2015年156卷5期

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In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model.

Pain

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Pain

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In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model.

Pain

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Pain

相关期刊

检索历史清除