DDIT3 and KAT2A Proteins Regulate TNFRSF10A and TNFRSF10B Expression in Endoplasmic Reticulum Stress-mediated Apoptosis in Human Lung Cancer Cells.

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作者： Tianliang,Li ^[1] ; Ling,Su ^[1] ; Yuanjiu,Lei ^[1] ; Xianfang,Liu ^[1] ; Yajing,Zhang ^[1] ; Xiangguo,Liu ^[2]

作者单位： From the Shandong University School of Life Sciences, Jinan 250100, China. ^[1] From the Shandong University School of Life Sciences, Jinan 250100, China xgliu@sdu.edu.cn. ^[2]

关键词 Apoptosis DIT3 Endoplasmic Reticulum Stress (ER Stress)Histone Acetylase KAT2A Lung Cancer TNFRSF10A TNFRSF10B Transcription Factor

主题词细胞凋亡(Apoptosis);细胞系, 肿瘤(Cell Line, Tumor);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);组蛋白酰基转移酶(Histone Acetyltransferases);人类(Humans);肺肿瘤(Lung Neoplasms);肿瘤蛋白质类(Neoplasm Proteins);受体, TNF相关凋亡诱导配体(Receptors, TNF-Related Apoptosis-Inducing Ligand);转录因子CHOP(Transcription Factor CHOP)

DOI 10.1074/jbc.M115.645333

PMID 25770212

发布时间 2021-02-05

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The Journal of biological chemistry

2015年290卷17期

11108-18页

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DDIT3 and KAT2A Proteins Regulate TNFRSF10A and TNFRSF10B Expression in Endoplasmic Reticulum Stress-mediated Apoptosis in Human Lung Cancer Cells.

The Journal of biological chemistry

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The Journal of biological chemistry

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DDIT3 and KAT2A Proteins Regulate TNFRSF10A and TNFRSF10B Expression in Endoplasmic Reticulum Stress-mediated Apoptosis in Human Lung Cancer Cells.

The Journal of biological chemistry

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The Journal of biological chemistry

相关期刊

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