换一批Alpha-synuclein overexpression negatively regulates insulin receptor substrate 1 by activating mTORC1/S6K1 signaling.
第一作者：
Shanshan,Gao
第一单位：
Center for Parkinson's Disease, Beijing Institute for Brain Disorders, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Department of Neurobiology, Capital Medical University, Beijing 100069, China.
作者：
医学主题词
动物(Animals);细胞系(Cell Line);基因表达(Gene Expression);人类(Humans);胰岛素受体底物蛋白质类(Insulin Receptor Substrate Proteins);小鼠, 近交C3H(Mice, Inbred C3H);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);多蛋白复合物(Multiprotein Complexes);蛋白质磷酸酶2(Protein Phosphatase 2);蛋白质稳定性(Protein Stability);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);核糖体蛋白质S6激酶类, 70-kDa(Ribosomal Protein S6 Kinases, 70-kDa);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);α突触核蛋白(alpha-Synuclein)
DOI
10.1016/j.biocel.2015.03.006
PMID
25813876
发布时间
2021-12-03
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