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Attenuating endogenous Fgfr2b ligands during bleomycin-induced lung fibrosis does not compromise murine lung repair.

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第一作者: BreAnne,MacKenzie
第一单位: German Center for Lung Research, Excellence Cluster Cardio-Pulmonary System, Universities of Giessen and Marburg Lung Center, Giessen, Hessen, Germany;
作者单位: German Center for Lung Research, Excellence Cluster Cardio-Pulmonary System, Universities of Giessen and Marburg Lung Center, Giessen, Hessen, Germany; [1] Developmental Biology Program, Division of Surgery, Saban Research Institute of Children's Hospital Los Angeles, University of Southern California Keck School of Medicine, Los Angeles, California; [2] School of Pharmacy, Wenzhou Medical College, China; [3] Comprehensive Pneumology Center, Ludwig Maximilians University, University Hospital Grosshadern, and Helmholtz Zentrum München, Munich, Bavaria, Germany; [4] Institute of Fundamental Medicine and Biology, Kazan Federal University, Kazan, Russia. [5] German Center for Lung Research, Excellence Cluster Cardio-Pulmonary System, Universities of Giessen and Marburg Lung Center, Giessen, Hessen, Germany; AGAPLESION Lung Clinic Waldhof-Elgershausen, Greifenstein, Germany; German Center for Lung Research, Germany; [6] German Center for Lung Research, Excellence Cluster Cardio-Pulmonary System, Universities of Giessen and Marburg Lung Center, Giessen, Hessen, Germany; Developmental Biology Program, Division of Surgery, Saban Research Institute of Children's Hospital Los Angeles, University of Southern California Keck School of Medicine, Los Angeles, California; Institute of Fundamental Medicine and Biology, Kazan Federal University, Kazan, Russia saverio.bellusci@innere.med.uni-giessen.de. [7]
DOI 10.1152/ajplung.00291.2014
PMID 25820524
发布时间 2023-07-10
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American journal of physiology. Lung cellular and molecular physiology

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