Cutting edge: Ubiquitin-specific protease 4 promotes Th17 cell function under inflammation by deubiquitinating and stabilizing RORγt.

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第一作者： Jing,Yang

第一单位： Key Laboratory of Molecular Virology and Immunology, Unit of Molecular Immunology, Institut Pasteur of Shanghai, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200025, China.

作者： Jing,Yang ^[1] ; Peng,Xu ^[2] ; Lei,Han ^[3] ; Zhixiang,Guo ^[2] ; Xiuwen,Wang ^[3] ; Zuojia,Chen ^[1] ; Jia,Nie ^[1] ; Shuying,Yin ^[1] ; Miranda,Piccioni ^[1] ; Andy,Tsun ^[1] ; Ling,Lv ^[4] ; Shenglin,Ge ^[5] ; Bin,Li ^[6]

作者单位： Key Laboratory of Molecular Virology and Immunology, Unit of Molecular Immunology, Institut Pasteur of Shanghai, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200025, China. ^[1] Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China; and. ^[2] Division of Rheumatology, Huashan Hospital, Fudan University, Shanghai 200040, China. ^[3] Division of Rheumatology, Huashan Hospital, Fudan University, Shanghai 200040, China huashanlvling@sina.com aydgsl@sina.com binli@sibs.ac.cn. ^[4] Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China; and huashanlvling@sina.com aydgsl@sina.com binli@sibs.ac.cn. ^[5] Key Laboratory of Molecular Virology and Immunology, Unit of Molecular Immunology, Institut Pasteur of Shanghai, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200025, China huashanlvling@sina.com aydgsl@sina.com binli@sibs.ac.cn. ^[6]