Length regulation of mechanosensitive stereocilia depends on very slow actin dynamics and filament-severing proteins.

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作者： Praveena,Narayanan ^[1] ; Paul,Chatterton ^[1] ; Akihiro,Ikeda ^[2] ; Sakae,Ikeda ^[2] ; David P,Corey ^[3] ; James M,Ervasti ^[1] ; Benjamin J,Perrin ^[4]

作者单位： Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, Minnesota 55455, USA. ^[1] Department of Medical Genetics, University of Wisconsin-Madison, Madison, Wisconsin 53706, USA. ^[2] Department of Neurobiology, Harvard Medical School and Howard Hughes Medical Institute, Boston, Massachusetts 02115, USA. ^[3] Department of Biology, Indiana University-Purdue University Indianapolis, Indianapolis, Indiana 46022, USA. ^[4]

主题词肌动蛋白类(Actins);动物(Animals);克隆, 分子(Cloning, Molecular);基因表达调控(Gene Expression Regulation);动力传导, 细胞(Mechanotransduction, Cellular);小鼠(Mice);Corti器(Organ of Corti)

DOI 10.1038/ncomms7855

PMID 25897778

发布时间 2018-11-13

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Nature communications

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Length regulation of mechanosensitive stereocilia depends on very slow actin dynamics and filament-severing proteins.

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Length regulation of mechanosensitive stereocilia depends on very slow actin dynamics and filament-severing proteins.

Nature communications

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Nature communications

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