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A REDD1/TXNIP pro-oxidant complex regulates ATG4B activity to control stress-induced autophagy and sustain exercise capacity.

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第一作者： Shuxi,Qiao

第一单位： 1] Massachusetts General Hospital Cancer Center, Boston, Massachusetts 02114, USA [2] Harvard Medical School, Boston, Massachusetts 02115, USA.

作者： Shuxi,Qiao ^[1] ; Michael,Dennis ^[2] ; Xiufeng,Song ^[1] ; Douangsone D,Vadysirisack ^[1] ; Devika,Salunke ^[2] ; Zachary,Nash ^[2] ; Zhifen,Yang ^[3] ; Marc,Liesa ^[4] ; Jun,Yoshioka ^[5] ; Shu-Ichi,Matsuzawa ^[3] ; Orian S,Shirihai ^[6] ; Richard T,Lee ^[5] ; John C,Reed ^[7] ; Leif W,Ellisen ^[1]

作者单位： 1] Massachusetts General Hospital Cancer Center, Boston, Massachusetts 02114, USA [2] Harvard Medical School, Boston, Massachusetts 02115, USA. ^[1] Massachusetts General Hospital Cancer Center, Boston, Massachusetts 02114, USA. ^[2] Sanford-Burnham Medical Research Institute, La Jolla, California 92037, USA. ^[3] Department of Medicine, Evans Center, Mitochondria ARC, Boston University School of Medicine, Boston, Massachusetts 02118, USA. ^[4] 1] Harvard Medical School, Boston, Massachusetts 02115, USA [2] Regenerative Medicine Center, Brigham and Women's Hospital, Boston, Massachusetts 02115, USA. ^[5] 1] Department of Medicine, Evans Center, Mitochondria ARC, Boston University School of Medicine, Boston, Massachusetts 02118, USA [2] Department of Clinical Biochemistry, Faculty of Medicine, Ben Gurion University of the Negev, Beer-Sheva 8410501, Israel. ^[6] 1] Sanford-Burnham Medical Research Institute, La Jolla, California 92037, USA [2] Roche Pharmaceutical Research and Early Development, Basel 4070, Switzerland. ^[7]