Altered Phenotype of β-Cells and Other Pancreatic Cell Lineages in Patients With Diffuse Congenital Hyperinsulinism in Infancy Caused by Mutations in the ATP-Sensitive K-Channel.
第一作者:
Rachel J,Salisbury
第一单位:
Centre for Endocrinology and Diabetes, Institute of Human Development, Faculty of Medical and Human Sciences, Manchester Academic Health Sciences Centre, The University of Manchester, Manchester, U.K.
作者:
医学主题词
病例对照研究(Case-Control Studies);细胞谱系(Cell Lineage);细胞增殖(Cell Proliferation);儿童(Child);儿童, 学龄前(Child, Preschool);细胞周期蛋白依赖激酶6(Cyclin-Dependent Kinase 6);周期素依赖激酶抑制剂p27(Cyclin-Dependent Kinase Inhibitor p27);胎儿(Fetus);胰高血糖素分泌细胞(Glucagon-Secreting Cells);同源盒结构域蛋白质类(Homeodomain Proteins);人类(Humans);婴儿(Infant);婴儿, 新生(Infant, Newborn);胰岛素(Insulin);胰岛素分泌细胞(Insulin-Secreting Cells);突变(Mutation);核蛋白质类(Nuclear Proteins);成对盒转录因子类(Paired Box Transcription Factors);钾通道, 内向整流(Potassium Channels, Inwardly Rectifying);生长抑素分泌细胞(Somatostatin-Secreting Cells);转录因子(Transcription Factors);斑马鱼蛋白质类(Zebrafish Proteins)
DOI
10.2337/db14-1202
PMID
25931474
发布时间
2025-05-29
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Diabetes
3182-8页
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