换一批
换一批Cannabinoid receptor 2 attenuates microglial accumulation and brain injury following germinal matrix hemorrhage via ERK dephosphorylation in vivo and in vitro.
第一作者：
Jun,Tang
第一单位：
Department of Neurosurgery, Southwest Hospital, Third Military Medical University, Chongqing, 400038, China.
作者：
主题词
动物(Animals);脑(Brain);脑水肿(Brain Edema);丁二烯类(Butadienes);大麻酚类(Cannabinoids);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);吲哚类(Indoles);颅内出血(Intracranial Hemorrhages);男(雄)性(Male);小神经胶质细胞(Microglia);神经变性(Nerve Degeneration);神经免疫调节(Neuroimmunomodulation);腈类(Nitriles);氧化性应激(Oxidative Stress);磷酰化(Phosphorylation);蛋白激酶抑制剂(Protein Kinase Inhibitors);随机分配(Random Allocation);受体, 大麻酚, CB2(Receptor, Cannabinoid, CB2);凝血酶(Thrombin);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.neuropharm.2015.04.028
PMID
25963415
发布时间
2018-12-11
- 浏览25
Neuropharmacology
424-33页
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