换一批Activating enhancer binding protein 2 epsilon (AP-2ε)-deficient mice exhibit increased matrix metalloproteinase 13 expression and progressive osteoarthritis development.
第一作者：
Stephan,Niebler
第一单位：
Institute of Biochemistry (Emil-Fischer-Center), Friedrich Alexander University Erlangen-Nürnberg, Fahrstrasse17, 91054, Erlangen, Germany. stephan.niebler@ukr.de.;Institute of Pathology, University Regensburg, Franz-Josef-Strauss-Allee 11, 93053, Regensburg, Germany. stephan.niebler@ukr.de.
作者：
医学主题词
动物(Animals);软骨, 关节(Cartilage, Articular);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);基因表达调控(Gene Expression Regulation);免疫组织化学(Immunohistochemistry);原位杂交(In Situ Hybridization);男(雄)性(Male);基质金属蛋白酶13(Matrix Metalloproteinase 13);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);显微镜检查, 电子, 透射(Microscopy, Electron, Transmission);骨关节炎(Osteoarthritis);骨生成(Osteogenesis);转录因子AP-2(Transcription Factor AP-2)
DOI
10.1186/s13075-015-0648-8
PMID
25964075
发布时间
2018-11-13
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