Ascorbic acid abrogates microparticle generation and vascular injuries associated with high-pressure exposure.

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作者： Ming,Yang ^[1] ; Veena M,Bhopale ^[1] ; Stephen R,Thom ^[2]

作者单位： Department of Emergency Medicine, University of Maryland, Baltimore, Maryland. ^[1] Department of Emergency Medicine, University of Maryland, Baltimore, Maryland sthom@smail.umaryland.edu. ^[2]

关键词 decompression microparticles neutrophil activation oxidative stress

主题词气压(Air Pressure);动物(Animals);膜联蛋白A5(Annexin A5);抗原(Antigens);抗氧化剂(Antioxidants);抗坏血酸(Ascorbic Acid);血管(Blood Vessels);毛细血管通透性(Capillary Permeability);细胞源性微粒(Cell-Derived Microparticles);减压病(Decompression Sickness);膳食, 素食者(Diet, Vegetarian);酶抑制剂(Enzyme Inhibitors);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);嗜中性白细胞激活(Neutrophil Activation);磷脂酰丝氨酸类(Phosphatidylserines)

DOI 10.1152/japplphysiol.00183.2015

PMID 25977448

发布时间 2015-07-03

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Journal of applied physiology (Bethesda, Md.

2015年119卷1期

77-82页

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Ascorbic acid abrogates microparticle generation and vascular injuries associated with high-pressure exposure.

Journal of applied physiology (Bethesda, Md.

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Journal of applied physiology (Bethesda, Md.

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Ascorbic acid abrogates microparticle generation and vascular injuries associated with high-pressure exposure.

Journal of applied physiology (Bethesda, Md.

相似文献

Journal of applied physiology (Bethesda, Md.

相关期刊

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