换一批
换一批Absence of nicotinic acetylcholine receptor α7 subunit amplifies inflammation and accelerates onset of fibrosis: an inflammatory kidney model.
第一作者：
Luan D,Truong
第一单位：
*Department of Pathology and Division of Nephrology, Department of Medicine, and The Methodist Hospital, Baylor College of Medicine, Houston, Texas, USA; and Department of Medicine, Division of Renal Diseases and Hypertension, University of Colorado Denver, Aurora, Colorado, USA.
作者：
主题词
动物(Animals);胶原(Collagen);细胞因子类(Cytokines);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);纤维蛋白(Fibrin);纤维化(Fibrosis);肾小球基底膜(Glomerular Basement Membrane);肾小球肾炎(Glomerulonephritis);炎症(Inflammation);肾(Kidney);巨噬细胞(Macrophages);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);蛋白质亚单位(Protein Subunits);金属蛋白酶2组织抑制剂(Tissue Inhibitor of Metalloproteinase-2);金属蛋白酶3组织抑制剂(Tissue Inhibitor of Metalloproteinase-3);转化生长因子β(Transforming Growth Factor beta)
DOI
10.1096/fj.14-262493
PMID
25985801
发布时间
2018-11-13
- 浏览7
FASEB journal
3558-70页
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