换一批
换一批Endothelial cell and podocyte autophagy synergistically protect from diabetes-induced glomerulosclerosis.
作者：
关键词
BUN, blood urea nitrogenCASP3, caspase 3, apoptosis-related cysteine peptidaseCdh5, cadherin 5DM, diabetes mellitusDN, diabetic nephropathyESRD, end-stage renal diseaseGBM, glomerular basement membraneGEC, glomerular endothelial cellsGFB, glomerular filtration barrierMAP1LC3A/B/LC3A/B), microtubule-associated protein 1 light chain 3 α/βMTOR, mechanistic target of rapamycinNphs2, nephrosis 2, podocinSQSTM1, sequestosome 1STZ, streptozotocinTEM, transmission electron microscopyTUBA, tubulinautophagydiabetic nephropathyendothelial cellspodocytesproteinuriasclerosisα, WT1, Wilms tumor 1
主题词
动物(Animals);细胞凋亡(Apoptosis);自噬(Autophagy);细胞, 培养的(Cells, Cultured);糖尿病肾病(Diabetic Nephropathies);内皮细胞(Endothelial Cells);基因缺失(Gene Deletion);肾小球滤过率(Glomerular Filtration Rate);葡萄糖(Glucose);整合酶类(Integrases);肾小球系膜细胞(Mesangial Cells);小鼠, 近交C57BL(Mice, Inbred C57BL);微管相关蛋白质类(Microtubule-Associated Proteins);表型(Phenotype);足细胞(Podocytes)
DOI
10.1080/15548627.2015.1049799
PMID
26039325
发布时间
2022-04-10
- 浏览137
Autophagy
1130-45页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
