Attenuation of the unfolded protein response and endoplasmic reticulum stress after mechanical unloading in dilated cardiomyopathy.
第一作者:
Estibaliz,Castillero
第一单位:
Division of Cardiothoracic Surgery, New York Presbyterian Hospital - College of Physicians and Surgeons of Columbia University, New York, New York;
作者:
主题词
细胞凋亡(Apoptosis);钙网蛋白(Calreticulin);心肌病, 扩张型(Cardiomyopathy, Dilated);病例对照研究(Case-Control Studies);细胞系(Cell Line);DNA结合蛋白质类(DNA-Binding Proteins);女(雌)性(Female);心室(Heart Ventricles);心脏辅助装置(Heart-Assist Devices);人类(Humans);男(雄)性(Male);中年人(Middle Aged);兰尼碱受体钙释放通道(Ryanodine Receptor Calcium Release Channel);肌浆网钙转运ATP酶类(Sarcoplasmic Reticulum Calcium-Transporting ATPases);转录因子(Transcription Factors);非折叠蛋白质应答(Unfolded Protein Response);eIF-2激酶(eIF-2 Kinase)
DOI
10.1152/ajpheart.00056.2015
PMID
26055788
发布时间
2020-09-30
- 浏览2
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文