Programmed cell death 5 mediates HDAC3 decay to promote genotoxic stress response.

导出 OA

翻译打印收藏纠错

作者： Hyo-Kyoung,Choi ^[1] ; Youngsok,Choi ^[2] ; Eun Sung,Park ^[3] ; Soo-Yeon,Park ^[1] ; Seung-Hyun,Lee ^[1] ; Jaesung,Seo ^[1] ; Mi-Hyeon,Jeong ^[1] ; Jae-Wook,Jeong ^[4] ; Jae-Ho,Jeong ^[5] ; Peter C W,Lee ^[6] ; Kyung-Chul,Choi ^[6] ; Ho-Geun,Yoon ^[1]

作者单位： Department of Biochemistry and Molecular Biology, Brain Korea 21 PLUS Project for Medical Sciences, Yonsei University College of Medicine, Seoul 120-752, Korea. ^[1] Department of Biomedical Science, CHA University, 335 Pangyo-ro, Bundang-gu, Seongnam-si, Gyeonggi-do 436-400, Korea. ^[2] Medical Convergence Research Institute, Yonsei University College of Medicine, Seoul 120-752, Korea. ^[3] Department of Obstetrics, Gynecology and Reproductive Biology, Michigan State University College of Human Medicine, Lansing, Michigan 49503, USA. ^[4] Department of Surgery, Yonsei University College of Medicine, Seoul 120-752, Korea. ^[5] Department of Biomedical Sciences, University of Ulsan College of Medicine, Seoul 138-736, Korea. ^[6]

主题词动物(Animals);凋亡调节蛋白质类(Apoptosis Regulatory Proteins);酪蛋白激酶Ⅱ(Casein Kinase II);细胞系, 肿瘤(Cell Line, Tumor);染色质免疫沉淀法(Chromatin Immunoprecipitation);DNA损伤(DNA Damage);成纤维细胞(Fibroblasts);基因表达谱(Gene Expression Profiling);HCT116细胞(HCT116 Cells);组蛋白脱乙酰基酶类(Histone Deacetylases);人类(Humans);免疫沉淀法(Immunoprecipitation);原位缺口末端标记(In Situ Nick-End Labeling);核胞浆转运蛋白类(Karyopherins);质谱分析法(Mass Spectrometry);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肿瘤蛋白质类(Neoplasm Proteins);肿瘤移植(Neoplasm Transplantation);磷酰化(Phosphorylation);预后(Prognosis);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);胃肿瘤(Stomach Neoplasms);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53)

DOI 10.1038/ncomms8390

PMID 26077467

发布时间 2021-11-08

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览2

Nature communications

2015年6卷

7390页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Programmed cell death 5 mediates HDAC3 decay to promote genotoxic stress response.

Nature communications

相似文献

Nature communications

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Programmed cell death 5 mediates HDAC3 decay to promote genotoxic stress response.

Nature communications

相似文献

Nature communications

相关期刊

检索历史清除