Over-expression of neurotrophin 3 in human aortic valves affected by calcific disease induces the osteogenic responses via the Trk-Akt pathway.

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作者： Qingzhou,Yao ^[1] ; Rui,Song ^[2] ; Lihua,Ao ^[3] ; Qiong,Zhan ^[4] ; Joseph C,Cleveland ^[3] ; Xiyong,Yu ^[5] ; David A,Fullerton ^[3] ; Xianzhong,Meng ^[6]

作者单位： Department of Surgery, University of Colorado Denver, Aurora, CO 80045, United States; Medical Research Center of Guangdong General Hospital, Guangdong Academy of Medical Sciences, Southern Medical University, Guangzhou 510080, China. ^[1] Department of Surgery, University of Colorado Denver, Aurora, CO 80045, United States; Departments of Pathophysiology, Southern Medical University, Guangzhou 510515, China. ^[2] Department of Surgery, University of Colorado Denver, Aurora, CO 80045, United States. ^[3] Department of Surgery, University of Colorado Denver, Aurora, CO 80045, United States; Departments of Cardiology of Nanfang Hospital, Southern Medical University, Guangzhou 510515, China. ^[4] Medical Research Center of Guangdong General Hospital, Guangdong Academy of Medical Sciences, Southern Medical University, Guangzhou 510080, China. Electronic address: yuxycn@aliyun.com. ^[5] Department of Surgery, University of Colorado Denver, Aurora, CO 80045, United States. Electronic address: Xianzhong.meng@ucdenver.edu. ^[6]

关键词 Akt Aortic valve Neurotrophin 3 Pro-osteogenic mediators Trk

DOI 10.1016/j.bbadis.2015.06.021

PMID 26122822

发布时间 2023-04-03

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