换一批
换一批Isoniazid prevents Nrf2 translocation by inhibiting ERK1 phosphorylation and induces oxidative stress and apoptosis.
第一作者：
Ajeet Kumar,Verma
第一单位：
PCS 103 Genotoxicity Lab, Division of Toxicology, CSIR-Central Drug Research Institute, B.S. 10/1, Sector 10, Jankipuram Extension, Sitapur Road, Lucknow 226031, India. Electronic address: ajeetsonicdri@gmail.com.
作者：
主题词
抗结核药(Antitubercular Agents);细胞凋亡(Apoptosis);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);基因表达谱(Gene Expression Profiling);基因表达调控(Gene Expression Regulation);热休克蛋白质类(Heat-Shock Proteins);肝细胞(Hepatocytes);人类(Humans);异烟肼(Isoniazid);代谢网络和途径(Metabolic Networks and Pathways);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);NF-E2相关因子2(NF-E2-Related Factor 2);氧化性应激(Oxidative Stress);磷酰化(Phosphorylation);蛋白质转运(Protein Transport);RNA, 信使(RNA, Messenger);活性氧(Reactive Oxygen Species);信号传导(Signal Transduction)
DOI
10.1016/j.redox.2015.06.020
PMID
26202867
发布时间
2019-03-09
- 浏览2
Redox biology
2015年6卷
80-92页
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