Inactivated Sendai virus induces apoptosis and autophagy via the PI3K/Akt/mTOR/p70S6K pathway in human non-small cell lung cancer cells.
作者:
主题词
抗肿瘤药(Antineoplastic Agents);细胞凋亡(Apoptosis);自噬(Autophagy);细胞系, 肿瘤(Cell Line, Tumor);顺铂(Cisplatin);抗药性, 肿瘤(Drug Resistance, Neoplasm);上皮细胞(Epithelial Cells);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);MAP激酶激酶4(MAP Kinase Kinase 4);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);呼吸道黏膜(Respiratory Mucosa);核糖体蛋白质S6激酶类, 70-kDa(Ribosomal Protein S6 Kinases, 70-kDa);仙台病毒(Sendai virus);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);病毒灭活(Virus Inactivation);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.bbrc.2015.07.130
PMID
26235873
发布时间
2022-03-30
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