Abolition of mitochondrial substrate-level phosphorylation by itaconic acid produced by LPS-induced Irg1 expression in cells of murine macrophage lineage.

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第一作者： Beáta,Németh

第一单位： *Department of Medical Biochemistry and Department of Physiology, Semmelweis University, Budapest, Hungary; and Lendület Neurobiochemistry Research Group, Lendület Inflammation Physiology Research Group, Laboratory for Neurobiochemistry, and Lendület Ion Channel Research Group, Hungarian Academy of Sciences, Budapest, Hungary.

作者： Beáta,Németh ^[1] ; Judit,Doczi ^[1] ; Dániel,Csete ^[1] ; Gergely,Kacso ^[1] ; Dora,Ravasz ^[1] ; Daniel,Adams ^[1] ; Gergely,Kiss ^[1] ; Adam M,Nagy ^[1] ; Gergo,Horvath ^[1] ; Laszlo,Tretter ^[1] ; Attila,Mócsai ^[1] ; Roland,Csépányi-Kömi ^[1] ; Iordan,Iordanov ^[1] ; Vera,Adam-Vizi ^[1] ; Christos,Chinopoulos ^[2]

作者单位： *Department of Medical Biochemistry and Department of Physiology, Semmelweis University, Budapest, Hungary; and Lendület Neurobiochemistry Research Group, Lendület Inflammation Physiology Research Group, Laboratory for Neurobiochemistry, and Lendület Ion Channel Research Group, Hungarian Academy of Sciences, Budapest, Hungary. ^[1] *Department of Medical Biochemistry and Department of Physiology, Semmelweis University, Budapest, Hungary; and Lendület Neurobiochemistry Research Group, Lendület Inflammation Physiology Research Group, Laboratory for Neurobiochemistry, and Lendület Ion Channel Research Group, Hungarian Academy of Sciences, Budapest, Hungary chinopoulos.christos@eok.sote.hu. ^[2]

关键词 cadA lipopolysaccharide succinate-CoA ligase