Antagonistic effects of IL-17 and D-resolvins on endothelial Del-1 expression through a GSK-3β-C/EBPβ pathway.
第一作者:
Tomoki,Maekawa
第一单位:
Department of Microbiology, Penn Dental Medicine, University of Pennsylvania, 240 S. 40th Street, Philadelphia, Pennsylvania 19104, USA.;Niigata University, Graduate School of Medical and Dental Sciences, Research Center for Advanced Oral Science, 2-5274 Gakkocho-dori, Chuo-ku, Niigata 951-8514, Japan.
作者:
医学主题词
牙槽骨质丢失(Alveolar Bone Loss);动物(Animals);CCAAT增强子结合蛋白β(CCAAT-Enhancer-Binding Protein-beta);钙结合蛋白质类(Calcium-Binding Proteins);载体蛋白质类(Carrier Proteins);细胞黏附分子(Cell Adhesion Molecules);染色质免疫沉淀法(Chromatin Immunoprecipitation);疾病模型, 动物(Disease Models, Animal);二十二碳六烯酸类(Docosahexaenoic Acids);减量调节(Down-Regulation);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);牙龈(Gingiva);糖原合成酶激酶3(Glycogen Synthase Kinase 3);人类(Humans);免疫印迹法(Immunoblotting);免疫沉淀法(Immunoprecipitation);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);白细胞介素17(Interleukin-17);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);牙周炎(Periodontitis);过氧化物酶(Peroxidase);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction)
DOI
10.1038/ncomms9272
PMID
26374165
发布时间
2026-01-22
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Nature communications
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