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GIT2 deficiency attenuates concanavalin A-induced hepatitis in mice.

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第一作者： Yu-E,Hao

第一单位： Southern Medical University, Guangzhou, Guangdong Province, China ; State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing 100850, China.

作者： Yu-E,Hao ^[1] ; Dong-Fang,He ^[2] ; Rong-Hua,Yin ^[3] ; Hui,Chen ^[3] ; Jian,Wang ^[3] ; Shao-Xia,Wang ^[3] ; Yi-Qun,Zhan ^[3] ; Chang-Hui,Ge ^[3] ; Chang-Yan,Li ^[3] ; Miao,Yu ^[3] ; Xiao-Ming,Yang ^[4]

作者单位： Southern Medical University, Guangzhou, Guangdong Province, China ; State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing 100850, China. ^[1] State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing 100850, China ; Anhui Medical University, Hefei 230032, Anhui Province, China. ^[2] State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing 100850, China. ^[3] Southern Medical University, Guangzhou, Guangdong Province, China ; State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing 100850, China ; Anhui Medical University, Hefei 230032, Anhui Province, China. ^[4]

关键词 Con A, concanavalin A FACS, fluorescence-activated cell sorting GFP, green fluorescent protein GIT2 GIT2, G protein-coupled receptor kinase interactor 2 Hepatitis Innate immunity Knock-out mice PMA, 4b-phorbol 12-myristate 13-acetate T cell activation TCR, T cell receptor

DOI 10.1016/j.fob.2015.08.005

PMID 26380813

发布时间 2018-11-13

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