Non-targeted metabolomics of <i>Brg1/Brm</i> double-mutant cardiomyocytes reveals a novel role for SWI/SNF complexes in metabolic homeostasis.

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第一作者： Ranjan,Banerjee

第一单位： University of North Carolina School of Medicine, Chapel Hill, NC, USA.

作者： Ranjan,Banerjee ^[1] ; Scott J,Bultman ^[2] ; Darcy,Holley ^[2] ; Carolyn,Hillhouse ^[3] ; James R,Bain ^[4] ; Christopher B,Newgard ^[4] ; Michael J,Muehlbauer ^[5] ; Monte S,Willis ^[6]

作者单位： University of North Carolina School of Medicine, Chapel Hill, NC, USA. ^[1] Department of Genetics, University of North Carolina, Chapel Hill, NC, USA. ^[2] Department of Pathology & Laboratory Medicine, University of North Carolina, Chapel Hill, NC, USA. ^[3] Sarah W. Stedman Nutrition and Metabolism Center, Duke Molecular Physiology Institute, Duke University Medical Center, Durham, NC, USA. Division of Endocrinology, Metabolism, and Nutrition, Department of Medicine, Duke University Medical Center, Durham, NC, USA. ^[4] Sarah W. Stedman Nutrition and Metabolism Center, Duke Molecular Physiology Institute, Duke University Medical Center, Durham, NC, USA. ^[5] Department of Pathology & Laboratory Medicine, University of North Carolina, Chapel Hill, NC, USA. McAllister Heart Institute, University of North Carolina, Chapel Hill, NC, USA. ^[6]

关键词 BRG1 BRM Cardiomyocyte Fatty acid Glucose Metabolomics SWI/SNF complex

DOI 10.1007/s11306-015-0786-7

PMID 26392817

发布时间 2019-11-20

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