Adaptor Protein-3-Mediated Trafficking of TLR2 Ligands Controls Specificity of Inflammatory Responses but Not Adaptor Complex Assembly.
作者:
主题词
衔接蛋白复合物3(Adaptor Protein Complex 3);动物(Animals);伯氏疏螺旋体(Borrelia burgdorferi);细胞, 培养的(Cells, Cultured);细胞因子类(Cytokines);宿主与病原体相互作用(Host-Pathogen Interactions);炎症介导素类(Inflammation Mediators);脂肽类(Lipopeptides);溶酶体(Lysosomes);巨噬细胞(Macrophages);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交C3H(Mice, Inbred C3H);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);显微镜检查, 共焦(Microscopy, Confocal);髓样分化因子88(Myeloid Differentiation Factor 88);吞噬体(Phagosomes);蛋白质转运(Protein Transport);受体, 白细胞介素(Receptors, Interleukin);受体, 白细胞介素1(Receptors, Interleukin-1);Toll样受体2(Toll-Like Receptor 2)
DOI
10.4049/jimmunol.1501268
PMID
26423153
发布时间
2021-08-10
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