换一批Nck1 deficiency improves pancreatic β cell survival to diabetes-relevant stresses by modulating PERK activation and signaling.
第一作者：
Lama,Yamani
第一单位：
Department of Medicine, McGill University and McGill University Health Centre-Research Institute, Montreal, Quebec H4A 3J1, Canada.
作者：
医学主题词
转录激活因子4(Activating Transcription Factor 4);主动转运, 细胞核(Active Transport, Cell Nucleus);衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);自噬(Autophagy);细胞存活(Cell Survival);细胞, 培养的(Cells, Cultured);糖尿病(Diabetes Mellitus);酶激活(Enzyme Activation);基因敲低技术(Gene Knockdown Techniques);胰岛素(Insulin);胰岛素分泌细胞(Insulin-Secreting Cells);NF-E2相关因子2(NF-E2-Related Factor 2);癌基因蛋白质类(Oncogene Proteins);信号传导(Signal Transduction);应激, 生理学(Stress, Physiological);转录因子CHOP(Transcription Factor CHOP);转录激活(Transcriptional Activation);eIF-2激酶(eIF-2 Kinase)
DOI
10.1016/j.cellsig.2015.09.016
PMID
26434994
发布时间
2018-12-17
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Cellular signalling
2555-67页
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