Novel cytogenic and neurovascular niches due to blood-brain barrier compromise in the chronic pain brain.

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作者： Maral,Tajerian ^[1] ; J David,Clark ^[2]

作者单位： Veterans Affairs Palo Alto Health Care System, 3801 Miranda Ave., Palo Alto, CA, 94304, USA. maral@stanford.edu. ^[1] Department of Anesthesiology, Stanford University School of Medicine, Stanford, CA, USA. maral@stanford.edu. ^[2] Palo Alto Institute of Research and Education, Palo Alto, CA, USA. maral@stanford.edu. ^[3] Veterans Affairs Palo Alto Health Care System, 3801 Miranda Ave., Palo Alto, CA, 94304, USA. djclark@stanford.edu. ^[4] Department of Anesthesiology, Stanford University School of Medicine, Stanford, CA, USA. djclark@stanford.edu. ^[5] Palo Alto Institute of Research and Education, Palo Alto, CA, USA. djclark@stanford.edu. ^[6]

主题词动物(Animals);血脑屏障(Blood-Brain Barrier);慢性病(Chronic Disease);人类(Humans);模型, 神经学(Models, Neurological)

DOI 10.1186/s12990-015-0066-6

PMID 26453186

发布时间 2018-11-13

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Molecular pain

2015年11卷

63页

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Novel cytogenic and neurovascular niches due to blood-brain barrier compromise in the chronic pain brain.

Molecular pain

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Novel cytogenic and neurovascular niches due to blood-brain barrier compromise in the chronic pain brain.

Molecular pain

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