Expression of activation-induced cytidine deaminase enhances the clearance of pneumococcal pneumonia: evidence of a subpopulation of protective anti-pneumococcal B1a cells.

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作者： Natsuo,Yamamoto ^[1] ; Steven M,Kerfoot ^[2] ; Andrew T,Hutchinson ^[1] ; Charles S,Dela Cruz ^[1] ; Naomi,Nakazawa ^[1] ; Marian,Szczepanik ^[2] ; Monika,Majewska-Szczepanik ^[1] ; Katarzyna,Nazimek ^[3] ; Noboru,Ohana ^[1] ; Krzysztof,Bryniarski ^[3] ; Tsutomu,Mori ^[1] ; Masamichi,Muramatsu ^[4] ; Keiji,Kanemitsu ^[2] ; Philip W,Askenase ^[1]

作者单位： Section of Allergy and Clinical Immunology, Department of Internal Medicine, Yale University School of Medicine, New Haven, CT, USA. ^[1] Department of Infection Control, Fukushima Medical University, Hikarigaoka, Japan. ^[2] Department of Medical Biology, Jagiellonian University Medical College, Krakow, Poland. ^[3] Department of Immunology, Jagiellonian University Medical College, Krakow, Poland. ^[4] Department of Molecular Genetics, Graduate School of Medical Science, Kanazawa University, Kanazawa, Japan. ^[5]

关键词 B1a cells IgM antibody activation-induced cytidine deaminase interleukin-13 invariant natural killer T cells pneumococcal pneumonia