GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels.
第一作者:
Priscila Y,Sato
第一单位:
Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
作者:
主题词
动物(Animals);细胞呼吸(Cell Respiration);脂肪酸类(Fatty Acids);G蛋白偶联受体激酶2(G-Protein-Coupled Receptor Kinase 2);小鼠, 转基因(Mice, Transgenic);线粒体, 心脏(Mitochondria, Heart);肌细胞, 心脏(Myocytes, Cardiac);氧耗量(Oxygen Consumption);应激, 生理学(Stress, Physiological);超氧化物类(Superoxides)
DOI
10.1016/j.yjmcc.2015.10.002
PMID
26506135
发布时间
2024-06-28
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