Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.

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作者： Bleranda,Zeka ^[1] ; Maria,Hastermann ^[1] ; Sonja,Hochmeister ^[2] ; Nikolaus,Kögl ^[1] ; Nathalie,Kaufmann ^[1] ; Kathrin,Schanda ^[3] ; Simone,Mader ^[3] ; Tatsuro,Misu ^[4] ; Paulus,Rommer ^[5] ; Kazuo,Fujihara ^[4] ; Zsolt,Illes ^[6] ; Fritz,Leutmezer ^[5] ; Douglas Kazutoshi,Sato ^[4] ; Ichiro,Nakashima ^[7] ; Markus,Reindl ^[4] ; Hans,Lassmann ^[3] ; Monika,Bradl ^[1]

作者单位： Department for Neuroimmunology, Center for Brain Research, Medical University Vienna, Spitalgasse 4, 1090, Vienna, Austria. ^[1] Department for Neurology, Medical University Graz, Auenbruggerplatz 22, 8036, Graz, Austria. ^[2] Clinical Department for Neurology, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria. ^[3] Department of Neurology, Tohoku University Graduate School of Medicine, 1-1 Seiryomachi, Aobaku, Sendai, 980-8574, Japan. ^[4] University Hospital for Neurology, Medical University Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. ^[5] Department of Neurology, University of Southern Denmark, Sdr Boulevard 29, Odense, 5000, Denmark. ^[6] Department of Neurology, Faculty of Medicine, University of Sao Paulo, Av. Dr. Arnaldo, 455-4th floor (sl 4110), 01246-903, São Paulo, Brazil. ^[7] Department for Neuroimmunology, Center for Brain Research, Medical University Vienna, Spitalgasse 4, 1090, Vienna, Austria. monika.bradl@meduniwien.ac.at. ^[8]

关键词 Aquaporin 4 CNS inflammation ENMO Neuromyelitis optica T cells