换一批Inhibitory effects of alternaramide on inflammatory mediator expression through TLR4-MyD88-mediated inhibition of NF-кB and MAPK pathway signaling in lipopolysaccharide-stimulated RAW264.7 and BV2 cells.
第一作者：
Wonmin,Ko
第一单位：
College of Pharmacy, Wonkwang University, Iksan 570-749, Republic of Korea.
作者：
医学主题词
链格孢属(Alternaria);动物(Animals);消炎药, 非甾类(Anti-Inflammatory Agents, Non-Steroidal);细胞系(Cell Line);缩肽类(Depsipeptides);剂量效应关系, 药物(Dose-Response Relationship, Drug);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);脂多糖类(Lipopolysaccharides);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);分子构象(Molecular Conformation);髓样分化因子88(Myeloid Differentiation Factor 88);NF-κB(NF-kappa B);构效关系(Structure-Activity Relationship);Toll样受体4(Toll-Like Receptor 4);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.cbi.2015.11.024
PMID
26620692
发布时间
2016-01-19
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