Aspirin Inhibits LPS-Induced Expression of PI3K/Akt, ERK, NF-κB, CX3CL1, and MMPs in Human Bronchial Epithelial Cells.
作者:
主题词
消炎药, 非甾类(Anti-Inflammatory Agents, Non-Steroidal);阿司匹林(Aspirin);支气管(Bronchi);细胞系(Cell Line);趋化因子CX3CL1(Chemokine CX3CL1);色酮类(Chromones);上皮细胞(Epithelial Cells);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);类黄酮物质(Flavonoids);人类(Humans);脂多糖类(Lipopolysaccharides);基质金属蛋白酶12(Matrix Metalloproteinase 12);基质金属蛋白酶7(Matrix Metalloproteinase 7);基质金属蛋白酶9(Matrix Metalloproteinase 9);吗啉类(Morpholines);NF-κB(NF-kappa B);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);吡咯烷类(Pyrrolidines);呼吸道黏膜(Respiratory Mucosa);硫代氨基甲酸酯类(Thiocarbamates)
DOI
10.1007/s10753-015-0289-8
PMID
26635114
发布时间
2019-12-10
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Inflammation
643-50页
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