AMPK interacts with β-catenin in the regulation of hepatocellular carcinoma cell proliferation and survival with selenium treatment.
第一作者:
Song Yi,Park
第一单位:
Department of Biological Sciences and Biotechnology, Hannam University Daedeok Valley Campus, Yuseong-gu, Daejeon 305-811, Republic of Korea.
作者:
主题词
AMP活化蛋白激酶类(AMP-Activated Protein Kinases);动物(Animals);癌, 肝细胞(Carcinoma, Hepatocellular);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);胞质溶胶(Cytosol);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);糖原合成酶激酶3(Glycogen Synthase Kinase 3);人类(Humans);肝肿瘤(Liver Neoplasms);小鼠(Mice);蛋白质结合(Protein Binding);RNA, 小分子干扰(RNA, Small Interfering);硒(Selenium);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays);β连环素(beta Catenin)
DOI
10.3892/or.2015.4519
PMID
26707164
发布时间
2017-11-16
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Oncology reports
1566-72页
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