论文期刊

取消

高级检索

检索历史清除

首页> Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine> A novel derivative of tetrandrine (H1) induces endoplasmic reticulum stress-mediated apoptosis and prosurvival autophagy in human non-small cell lung cancer cells.

A novel derivative of tetrandrine (H1) induces endoplasmic reticulum stress-mediated apoptosis and prosurvival autophagy in human non-small cell lung cancer cells.

导出原文传递

翻译打印收藏纠错

作者： Yidan,Lin ^[1] ; Yu,Wang ^[2] ; Xianfang,Liu ^[3] ; Jiamei,Yan ^[2] ; Ling,Su ^[2] ; Xiangguo,Liu ^[4]

作者单位： The Thoracic Surgery Department of West China Hospital, West China Medical School of Sichuan University, Chengdu, China. ^[1] The Provincial Key Laboratory of Animal Cells and Developmental Biology, School of Life Sciences, Shandong University, Rm 103, South BLDG, 27 Shanda South Road, Jinan, 250100, China. ^[2] The Department of Otolaryngology Head and Neck Surgery, Provincial Hospital Affiliated to Shandong University, Jinan, China. ^[3] The Provincial Key Laboratory of Animal Cells and Developmental Biology, School of Life Sciences, Shandong University, Rm 103, South BLDG, 27 Shanda South Road, Jinan, 250100, China. xgliu@sdu.edu.cn. ^[4]

关键词 Apoptosis Autophagy CHOP DR5 ER stress Tetrandrine c-FLIP

主题词抗肿瘤药(Antineoplastic Agents);细胞凋亡(Apoptosis);苄异喹啉类(Benzylisoquinolines);印迹法, 蛋白质(Blotting, Western);癌, 非小细胞肺(Carcinoma, Non-Small-Cell Lung);细胞系, 肿瘤(Cell Line, Tumor);基因敲低技术(Gene Knockdown Techniques);人类(Humans);肺肿瘤(Lung Neoplasms);受体, TNF相关凋亡诱导配体(Receptors, TNF-Related Apoptosis-Inducing Ligand)

DOI 10.1007/s13277-016-4950-0

PMID 26846103

发布时间 2021-12-03

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览6

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Tumour biology

2016年37卷8期

10403-13页

相似文献

中文期刊
外文期刊
学位论文
会议论文

特别提示：本网站仅提供医学学术资源服务，不销售任何药品和器械，有关药品和器械的销售信息，请查阅其他网站。

客服热线：4000-115-888 转3 (周一至周五：8:00至17:00)
|
客服邮箱：yiyao@wanfangdata.com.cn

违法和不良信息举报电话：4000-115-888，举报邮箱：problem@wanfangdata.com.cn，举报专区

官方微信

万方医学小程序

关于我们网站地图知识产权

北京万方数据股份有限公司

万方数据电子出版社

京ICP证010071号

京公网安备11010802020237号

京ICP备08100800号-1

违法和不良信息举报电话：4000-115-888，举报邮箱：problem@wanfangdata.com.cn，举报专区

new翻译充值订阅收藏移动端

官方微信

万方医学小程序

调查问卷